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UCI Authors in PubMed  |
Waxman K
California College of Medicine, University of California Irvine, Medical Center, Orange.
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Postoperative multiple organ failure.
Crit Care Clin. 1987 Apr;3(2):429-40
Cellular damage is the pathophysiologic basis for the postoperative multiple organ failure syndrome. This damage may be caused by pre- and intraoperative shock. Postoperative organ failure is manifested when cellular repair does not occur. Three factors may contribute to this progression to multiple organ failure: inadequate resuscitation, malnutrition, and sepsis.
PMID: 3332208
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Fairshter RD,Williams JH
Department of Medicine, University of California Irvine, Orange.
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Pulmonary physiology in the postoperative period.
Crit Care Clin. 1987 Apr;3(2):287-306
After upper abdominal or thoracic surgery, loss of lung volume, manifested most importantly by reduced FRC, and abnormal gas exchange, manifested by postoperative reduction in PaO2, are the rule rather than the exception. These physiologic alterations in lung function occur in the absence of superimposed complications such as pneumonia and/or lobar atelectasis; the usual physiologic alterations do, in fact, predispose to pulmonary complications (approximate 20 per cent incidence) which, when present, further compromise lung function. The mechanisms of postoperative impairment in lung function are multiple, interactive, and at the present, incompletely understood. An attempt to summarize the more important causes of impaired postoperative lung function is shown in Figure 6.
PMID: 3332201
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Waxman K
California College of Medicine, University of California Irvine Medical Center, Orange.
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Hemodynamic and metabolic changes during and following operation.
Crit Care Clin. 1987 Apr;3(2):241-50
During anesthesia and operation, there appears to be a common state wherein oxygen consumption is inadequate to meet intraoperative metabolic requirements. Although there is insufficient information to fully explain this problem, decreased intraoperative levels of cardiac output and oxygen delivery, altered intraoperative oxygen transport at the microcirculatory and cellular levels, and altered delivery of oxidative substrate are possible contributors. Anesthesia and operation thus appear to produce a physiologic pattern similar to other shock states. Postoperative physiologic changes include increased cardiac output and increased oxygen delivery, necessary to support increased oxygen consumption. These postoperative changes may represent compensatory physiologic responses to preoperative and intraoperative oxidative and metabolic deficits. There are also significant energy needs of the healing surgical wound. These additional energy requirements for wound healing add to the degree of increase in metabolism necessary for recovery from operation. The postoperative increase in oxygen consumption appears to be essential to reverse intraoperative deficits, and has been reported to have survival value. Clinically, a major goal of postoperative therapy should thus be to support these necessary physiologic compensations. Titration of postoperative therapy should therefore not be to "normal" physiologic endpoints, but to the supernormal cardiac output and oxygen transport necessary for recovery in postoperative patients.
PMID: 3332198
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Cole-Beuglet C,Aufrichtig D,Cohen A,Harrison L,Miller EI,Crade M
Department of Radiological Sciences, University of California Irvine Medical Center.
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Ultrasound case of the day. Twin pregnancy, intrauterine death of one twin with disseminated intravascular coagulation resulting in the development of a cerebral infarct in the surviving twin.
Radiographics. 1987 Mar;7(2):389-94
PMID: 3329360
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Asch RH,Balmaceda JP,Ord T,Borrero C,Rodriguez Rigau LJ,Rojas FJ
UCI/AMI Center for Reproductive Health, Orange.
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Gamete intra fallopian transfer (GIFT) and oocyte donation--a novel treatment for infertility in premature ovarian failure.
Gynecol Endocrinol. 1987 Mar;1(1):105-11
PMID: 2459900
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Burney LR,Dumars KW,Chea CS,Nguyen HD,Bustillo AM
University of California-Irvine Medical Center, Orange 92668.
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The Southeast Asian refugee: the impact of cultural variation on the genetic counseling process.
Birth Defects Orig Artic Ser. 1987;23(6):239-44
PMID: 3435766
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Achauer BM,Vander Kam VM
University of California Irvine Medical Center, Division of Plastic Surgery, Orange 92668.
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Argon laser treatment of telangiectasia of the face and neck: 5 years' experience.
Lasers Surg Med. 1987;7(6):495-8
This study documents the treatment and long-term follow-up of telangiectasias of the face and neck with the argon laser. This study concurs with other examiners that treatment is effective with minimal complications and satisfactory long-term results.
PMID: 3431325
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Kaufman E
Department of Psychiatry, University of California Irvine Medical Center, Orange 92668.
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A contemporary approach to the family treatment of substance abuse disorders.
Am J Drug Alcohol Abuse. 1986;12(3):199-211
A contemporary approach to the family treatment of substance abuse is presented. This approach involves developing a method for establishing and maintaining a substance-free state, a workable system of family therapy, sufficient attention to the family after substance abuse has stopped, and an individualized, integrated approach which takes into consideration a variety of family factors.
PMID: 3503566
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Wasmuth JJ,Hill JM,Vock LS
Department of Biological Chemistry, California College of Medicine, University of California-Irvine 92717.
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Identification and characterization of a third complementation group of emetine-resistant Chinese hamster cell mutants.
Mol Cell Biol. 1981 Jan;1(1):58-65
We have isolated emetine-resistant cell lines from Chinese hamster peritoneal fibroblasts and have shown that they represent a third distinct class or complementation group of emetine-resistant mutants, as determined by three different criteria. These mutants, like those belonging to the two other complementation groups we have previously defined, which were isolated from Chinese hamster lung and Chinese hamster ovary cells, have alterations that directly affect the protein biosynthetic machinery. So far, there is absolute cell line specificity with respect to the three complementation groups, in that all the emetine-resistant mutants we have isolated from Chinese hamster lung cells belong to one complementation group, all those we have isolated from Chinese hamster ovary cells belong to a second complementation group, and all those isolated from Chinese hamster peritoneal cells belong to a third complementation group. Thus, in cultured Chinese hamster cells, mutations in at least three different loci, designated emtA, emtB, and emtC, encoding for different components of the protein biosynthetic machinery, can give rise to the emetine-resistant phenotype.
PMID: 6965094
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Gerard RW
UNIVERSITY OF CALIFORNIA (IRVINE).
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COMPUTER-ASSISTED LEARNING: INTRODUCTION AND GENERAL CONSIDERATIONS.
Proc Natl Acad Sci U S A. 1969 Jul;63(3):573-579
PMID: 16591765
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